Web24 de set. de 2024 · Prinz M, Erny D, Hagemeyer N (2024) Ontogeny and homeostasis of CNS myeloid cells. Nat Immunol 18:385–392. CrossRef CAS Google Scholar Giulian D, Li J, Bartel S, Broker J, Li X, Kirkpatrick JB (1995) Cell surface morphology identifies microglia as a distinct class of mononuclear phagocyte. J Neurosci 15:7712–7726 Web3 de dez. de 2024 · STEM CELLS is read and written by clinical and basic scientists whose expertise encompasses the rapidly expanding fields of stem and progenitor cell biology. STEM CELLS welcomes original articles and concise reviews describing basic laboratory investigations of stem cells and the translation of their clinical aspects of characterization …
Autophagy Pathways in CNS Myeloid Cell Immune Functions
Web26 de abr. de 2024 · Models of cellular ontogeny, activation, differentiation, and tissue-specific functions of myeloid cells have been revisited during the last years with sur … Annu Rev Immunol . 2024 Apr 26;37:269-293. doi: … Web1 de mar. de 2024 · Myeloid cells in the central nervous system (CNS) represent a heterogeneous class of innate immune cells that contribute to the maintenance of tissue … ethicon mx69
Bruton tyrosine kinase inhibitors for multiple sclerosis Nature ...
WebThe CNS accommodates a diverse myeloid immune cell compartment that maintains CNS homeostasis in the steady state while contributing to tissue injury during infectious, autoimmune, and neurodegenerative disease conditions. Autophagy and autophagy proteins play fundamental roles in myeloid cell–related immune functions. Many of these … Web16 de out. de 2024 · Yet their unique ontogeny and distinguishing features endowed by the privileged nature of their environment have set them apart from other myeloid cells in the body. Microglia spectrum of activity enables their participation in the maintenance of homeostasis and disease processes via their interaction with CNS cells as well as the … WebOntogeny and homeostasis of CNS myeloid cells. Nat. Immunol. 2024; 18: 385-392. Crossref; PubMed; Scopus (255) Google Scholar]. By contrast, CX3CR1-expressing myeloid cells that are recruited to the brain during inflammatory insults are short-lived and renew from CX3CR1-negative progenitors located in bone marrow [27. ethicon mesh nj wilentz